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Download Acute Myelogenous Leukemia - A Medical Dictionary, by Icon Health Publications PDF

By Icon Health Publications

This can be a 3-in-1 reference e-book. It provides an entire clinical dictionary protecting hundreds and hundreds of phrases and expressions in relation to acute myelogenous leukemia. It additionally provides huge lists of bibliographic citations. ultimately, it offers details to clients on find out how to replace their wisdom utilizing quite a few web assets. The publication is designed for physicians, scientific scholars getting ready for Board examinations, clinical researchers, and sufferers who are looking to get to grips with study devoted to acute myelogenous leukemia. in case your time is efficacious, this booklet is for you. First, you won't waste time looking the web whereas lacking loads of suitable details. moment, the e-book additionally saves you time indexing and defining entries. eventually, you won't waste time and cash printing enormous quantities of web content.

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3. Generate and characterize mouse models for FPD/AML that mimic the hematopoietic progenitor defects, platelet defects, and propensity for AML. ; Associate Professor; Dana-Farber Cancer Institute 44 Binney St Boston, Ma 02115 Timing: Fiscal Year 2002; Project Start 01-AUG-2002; Project End 31-MAR-2003 Summary: (provided by applicant): Acute myelogenous leukemia (AML) is characterized by chromosomal translocations or point mutations that involve transcription factors, such as PML/RARa, AML1/ETO and C/EBPa.

Finally, the normal in vivo functions of NUP98 and HOXA9 will be studied by examining phenotypic effects of loss- or gain-of-function mutations in mice. These studies should further our understanding of the molecular mechanism of oncogenesis in an expanding subgroup o AML patients with translocations that link nucleoporins to nuclear proteins. ; Assistant Professor; George Williams Hooper Foundation; University of California San Francisco 500 Parnassus Ave San Francisco, Ca 941222747 Timing: Fiscal Year 2002; Project Start 19-FEB-1999; Project End 31-JAN-2004 Summary: A differentiation block is a cardinal attribute of nearly all human cancers and leukemias.

Further, these mice will be bred onto a BXH2 genetic background to identify mutations that cooperate with NUP98-HOXA9 to induce AML. Finally, the normal in vivo functions of NUP98 and HOXA9 will be studied by examining phenotypic effects of loss- or gain-of-function mutations in mice. These studies should further our understanding of the molecular mechanism of oncogenesis in an expanding subgroup o AML patients with translocations that link nucleoporins to nuclear proteins. ; Assistant Professor; George Williams Hooper Foundation; University of California San Francisco 500 Parnassus Ave San Francisco, Ca 941222747 Timing: Fiscal Year 2002; Project Start 19-FEB-1999; Project End 31-JAN-2004 Summary: A differentiation block is a cardinal attribute of nearly all human cancers and leukemias.

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